Diabetic foot syndrome


Diabetic foot syndrome is a set of pathological changes that appear on the lower extremities in patients suffering from diabetes.

Diabetic foot syndrome

is a set of pathological changes that appear on the lower extremities in patients suffering from diabetes.
Diabetic foot syndrome is caused by changes in the blood vessels (ischemia) and damage to the nerve fibres (neuropathy). Deterioration of the vascular supply and nerve supply in the diabetic foot usually progresses simultaneously, but only one of these changes dominates. The dominant factor determines the type of the condition: ischemic diabetic foot syndrome or neuropathic diabetic foot syndrome.

Peripheral neuropathy or peripheral nerve inflammation

is one of the complications of prolonged hyperglycaemia. It affects both somatic and autonomic nerve cell extensions in both legs. The patient experiences impaired perception of pain, temperature and touch in the affected leg; because they do not feel pain from injury or pressure, skin damage and ulcers often develop and go unnoticed by the patient. As a result, they do not seek help immediately. If an ulcer is detected late, the lesion may be too advanced to be treated quickly.

Motor neuropathy

causes a loss of strength in the muscles and tendons of the foot, and subsequently leads to joint destruction. This in turn causes the body weight to be concentrated in several small areas of the sole of the foot, which results in the formation of calluses. The final stage of the condition is foot deformity.

Angiopathy or vascular injury

impaired blood supply characterised by abnormal flow in the arteries of the lower legs, particularly the tibial and fibular arteries. Uncontrolled diabetes promotes the development of arterial disease. Effective management of blood glucose levels is both a prevention strategy and a prerequisite for the healing process to begin.

There are several types of ulcers:

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